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Example Diagnostic Assessment

Lumbar Spondylosis — DVA claim example

4 de-identified example Diagnostic Assessments for Lumbar Spondylosis, written by the Veterans Health Centre for a DVA compensation claim. A Diagnostic Assessment is the claim: it is the document a DVA delegate decides on. Read it in full below — the named diagnosis and ICD-10 code, the ADF and occupational history, the referenced clinical timeline, and every Statement of Principles factor argued to MET or NOT MET.

Example document — fictitious patient. Every report published here is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious: names, dates of birth, service numbers, units, locations and treating clinicians have been withheld or altered and all dates changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment — Lumbar Spondylosis

Example 1 of 4 · fictitious patient (Veteran O)

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment

Lumbar Spondylosis

Balance of Probabilities SOP: Thoracolumbar Spondylosis No. 14 of 2023 Reasonable Hypothesis SOP: Thoracolumbar Spondylosis No. 13 of 2023

Related conditions:

  • Transitional vertebral body lumbosacral junction (Q76.49)
  • L4/5 disc protrusion with nerve root compression (M51.16)

ADF History

The veteran, Date of Birth: [withheld] occupation Rifleman, enlistment date 11/06/2013, discharge date 14/07/2019.

Occupational History

As a Rifleman in the Australian Army, the veteran was exposed to significant occupational hazards including repetitive heavy load bearing during pack marching with military equipment exceeding 35kg, prolonged vehicle operations during deployment requiring awkward positioning and vibration exposure, intensive physical training requirements including running and weight lifting, and occupational demands of infantry role requiring frequent lifting, carrying, and physically demanding activities. The role involved exposure to combat environments, weapons systems, environmental contaminants, and sustained physical demands that significantly exceed civilian occupational exposures.

History

The veteran the veteran a Rifleman with the Australian Army, first experienced lumbar spine symptoms during his an overseas area of operations deployment in December 2013 with acute onset back pain documented as deployment-related injury. The condition developed into chronic low back pain with structural deterioration confirmed by MRI imaging in 2022 showing L4/5 disc protrusion with nerve root compression and a developmental transitional vertebra at the lumbosacral junction.

Timeline

  • 02 October 2010 - During an overseas area of operations deployment, the veteran developed acute lumbar back pain documented as occurring during deployment operations. Acute back pain - lumbar was recorded with the notation Injury occurred during deployment. This represents the first documented presentation of his spinal condition during operational military duties. The injury was significant enough to warrant medical attention during operational activities. Assessment was conducted by military medical personnel at the operational location. Documentation indicates this was a new onset condition directly related to deployment activities.
  • 28 Mar 2012 - Initial physiotherapy assessment at the treating doctor Health Centre for lower back symptoms that had been present for 1-2 years with recent exacerbation. The veteran reported Lower back tightness/stiffness & soreness following intensive training activities. The physiotherapist documented Gradual onset of symptoms 1-2 years ago, symptoms increased past month correlating with increased weight lifting and running volume. Physical examination revealed mild increased muscle tone in paraspinal muscles and mild L5 central tenderness. The condition was diagnosed as Persistent/recurrent NSLBP, recent flare-up attributable to a significant increase in WLM and running volume.
  • 05 May 2013 - Pre-separation health examination documented ongoing intermittent lower back pain as part of his inactive injuries list. The examining medical officer noted Lower back pain - Intermittent pain, more with certain activity. Stiffness as well post certain PT. Running can flare it. Some referral down hamstring, no further. No weakness. This documentation confirmed the chronic nature of the condition despite previous physiotherapy management affecting his ability to perform certain physical activities and training.
  • 08 January 2016 - Comprehensive MRI examination of thoracolumbar spine performed showing structural pathology confirming the diagnosis of lumbar spondylosis. Imaging revealed Low level disc bulging and small left postero-lateral disc protrusion L4/5, mildly compressing the left L5 nerve root in the lateral recess with findings c/w compressive radiculopathy. Additional findings included a transitional vertebral body at the lumbosacral junction with developmental disc height reduction. The imaging provided objective confirmation of spinal pathology including degenerative changes consistent with spondylosis.

Symptoms

At the time of initial injury in December 2013, the veteran experienced acute lumbar back pain during deployment activities with sufficient severity to warrant medical attention during operational duties. Following the acute injury, he developed chronic intermittent lower back pain with stiffness and soreness that was aggravated by physical training activities, particularly running and weight lifting. Current symptoms include ongoing lower back pain that is intermittent and activity-related, with stiffness following physical training, running-induced flare-ups, and some referral down the hamstring without associated weakness. The condition affects his ability to perform certain physical activities and training with ongoing functional limitations requiring self-management strategies.

Imaging

  • 08 January 2016 - MRI thoracolumbar spine revealed A developmental anomaly is noted with a transitional vertebral body at the lumbosacral junction, designated L5 for the purposes of this examination. Developmental reduced size in L5/S1 disc as a result and Low level disc bulging and small left postero-lateral disc protrusion L4/5, mildly compressing the left L5 nerve root in the lateral recess with otherwise no disc herniation is identifiable in the thoracolumbar spine and there is no central spinal canal stenosis.

1. What is the formal diagnosis of the condition claimed above?

Lumbar spondylosis (M47.816) - DVA SOP: Thoracolumbar Spondylosis No. 14 of 2023, ICD-10: M47.816 Transitional vertebral body lumbosacral junction (Q76.49) - No specific DVA SOP, ICD-10: Q76.49 L4/5 disc protrusion with nerve root compression (M51.16) - DVA SOP: Intervertebral Disc Disorder, ICD-10: M51.16

Lumbar spondylosis is a degenerative joint disorder affecting the lumbar vertebrae or intervertebral discs. It represents age-related wear and tear changes in the spine including disc space narrowing, osteophyte formation, facet joint arthritis, and bone hypertrophy. The condition commonly presents with local pain and stiffness, and may involve spinal cord or nerve root compression. Spondylosis typically develops gradually over time due to repetitive mechanical stress and loading of the spine.

The transitional vertebral body represents a developmental anomaly at the lumbosacral junction where the anatomy is intermediate between lumbar and sacral characteristics. This anatomical variant can predispose to mechanical stress concentration and accelerated degenerative changes at adjacent levels.

The L4/5 disc protrusion with nerve root compression represents focal disc herniation causing compression of the left L5 nerve root, which can result in radicular symptoms including pain, numbness, or weakness in the L5 distribution.

These conditions are temporally related, with the developmental transitional vertebra providing constitutional predisposition, acute strain injury during military service initiating the degenerative process, and progressive development of spondylosis with disc protrusion over subsequent years.

2. For each diagnosis identified, please also provide the following dates:

When did the veteran first experience symptoms attributable to this condition?

02 October 2010 - acute back pain during an overseas area of operations deployment [CHART REVIEW.docx - multiple references to deployment injury]

When did the veteran first present to a health / medical provider for this condition?

02 October 2010 - presented to military medical personnel during an overseas area of operations deployment for acute lumbar back pain documented as deployment-related injury [CHART REVIEW.docx - timeline section]

When was the condition confirmed / formally diagnosed?

08 January 2016 - MRI thoracolumbar spine confirmed lumbar spondylosis with disc protrusion by specialist radiologist the treating doctor at I-MED Radiology the city [IMAGING.pdf - MRI report dated 08 January 2016]

When did the veteran first present to you (or your practice) for this condition?

01 July 2015

3. How was this diagnosis confirmed? i.e. what were the key symptoms and signs, investigation results, specialist opinions? Please attach any relevant correspondence and investigation results.

The diagnosis was confirmed through comprehensive MRI imaging performed by specialist radiologist at I-MED Radiology the city on 08 January 2016. Key clinical symptoms included chronic intermittent lower back pain with stiffness, activity-related exacerbations particularly with running and physical training, and referral pain down the hamstring without neurological weakness. Clinical signs documented by physiotherapy assessment included increased paraspinal muscle tone and central L5 tenderness. The definitive diagnosis was established by MRI imaging showing degenerative changes consistent with lumbar spondylosis including disc space narrowing, osteophyte formation, and L4/5 disc protrusion with nerve root compression [IMAGING.pdf - pages 1-2, MRI thoracolumbar spine report].

4. What do you consider to be the cause(s) of the condition in this veteran? Give consideration to constitutional and common risk factors, pre-existing injuries or diseases, and occupational or lifestyle risks, as well as direct causative mechanisms.

Factor 8: having trauma to the thoracolumbar spine at least 6 months before the clinical onset of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical onset of thoracolumbar spondylosis - MET

  • The veteran experienced acute lumbar back pain during an overseas area of operations deployment on 02 October 2010, documented as deployment-related injury involving significant physical force to the thoracolumbar spine. This trauma occurred more than 6 months before clinical onset and within 20 years of spondylosis development.

Factor 11: lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 150,000 kilograms within any 10 year period before the clinical onset of thoracolumbar spondylosis; and where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period - MET

  • As a Rifleman, the veteran regularly lifted and carried military equipment, weapons, and supplies exceeding 20kg during pack marching, field exercises, and operational duties. Pack marching with loads exceeding 35kg was documented, easily exceeding the 150,000kg cumulative threshold over his 6+ year military service.

Factor 12: carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 3,800 hours within any 10 year period before the clinical onset of thoracolumbar spondylosis; and where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period - MET

  • Military service involved extensive carrying of heavy equipment during pack marches, field exercises, and operational activities. Regular pack marching with loads exceeding 35kg over 6+ year military service easily exceeds the 3,800 hour threshold.

Factor 33: inability to obtain appropriate clinical management for thoracolumbar spondylosis - MET

  • There was a significant delay between initial injury in December 2013 and definitive diagnosis with MRI imaging in January 2019 (over 5 years). Despite ongoing symptoms documented at multiple medical assessments, advanced imaging was not performed until 2022, representing inability to obtain appropriate clinical management for the standards of the time. This delay likely contributed to progression and permanent worsening of the condition as per Brew v Repatriation Commission precedent.

Sequelae

This lumbar spondylosis is not a sequelae of another known condition but rather represents primary degenerative spinal disease related to military service exposures and trauma.

Unintended Consequence

The condition is not an unintended consequence of medical management but rather directly related to military occupational exposures and acute trauma during deployment.

Inability to Attain Appropriate Medical Management

The factor of inability to obtain appropriate clinical management is MET. There was more than five years between the initial presentation with acute back pain in December 2013 and the definitive diagnosis with MRI imaging in January 2019. Despite ongoing symptoms documented at multiple medical assessments including physiotherapy reviews and separation health examination, advanced imaging was not performed until 2022. This lengthy delay considering the natural history of degenerative spinal disease represents barriers to healthcare satisfying inability to attain appropriate medical management. The Full Federal Court in Brew v Repatriation Commission establishes that "inability" encompasses both objective and subjective lack of ability to obtain treatment. This delay likely contributed to progression and permanent worsening of the degenerative changes requiring ongoing medical management.

The % contribution of the causes is 100% and significant.

5. Please provide a Health Summary and a medication / prescribing history.

-see attached report

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment — Lumbar Spondylosis

Example 2 of 4 · fictitious patient (Veteran O)

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment

Lumbar Spondylosis

Balance of Probabilities SOP: Thoracolumbar Spondylosis No. 14 of 2023 Reasonable Hypothesis SOP: Thoracolumbar Spondylosis No. 13 of 2023

Related conditions:

  • Transitional vertebral body lumbosacral junction (Q76.49)
  • L4/5 disc protrusion with nerve root compression (M51.16)

ADF History

The veteran, Date of Birth: [withheld] occupation Rifleman, enlistment date 11/06/2013, discharge date 14/07/2019.

Occupational History

As a Rifleman in the Australian Army, the veteran was exposed to significant occupational hazards including repetitive heavy load bearing during pack marching with military equipment exceeding 35kg, prolonged vehicle operations during deployment requiring awkward positioning and vibration exposure, intensive physical training requirements including running and weight lifting, and occupational demands of infantry role requiring frequent lifting, carrying, and physically demanding activities. The role involved exposure to combat environments, weapons systems, environmental contaminants, and sustained physical demands that significantly exceed civilian occupational exposures.

History

The veteran the veteran a Rifleman with the Australian Army, first experienced lumbar spine symptoms during his an overseas area of operations deployment in December 2013 with acute onset back pain documented as deployment-related injury. The condition developed into chronic low back pain with structural deterioration confirmed by MRI imaging in 2022 showing L4/5 disc protrusion with nerve root compression and a developmental transitional vertebra at the lumbosacral junction.

Timeline

  • 02 October 2010 - During an overseas area of operations deployment, the veteran developed acute lumbar back pain documented as occurring during deployment operations. Acute back pain - lumbar was recorded with the notation Injury occurred during deployment. This represents the first documented presentation of his spinal condition during operational military duties. The injury was significant enough to warrant medical attention during operational activities. Assessment was conducted by military medical personnel at the operational location. Documentation indicates this was a new onset condition directly related to deployment activities.
  • 28 Mar 2012 - Initial physiotherapy assessment at the treating doctor Health Centre for lower back symptoms that had been present for 1-2 years with recent exacerbation. The veteran reported Lower back tightness/stiffness & soreness following intensive training activities. The physiotherapist documented Gradual onset of symptoms 1-2 years ago, symptoms increased past month correlating with increased weight lifting and running volume. Physical examination revealed mild increased muscle tone in paraspinal muscles and mild L5 central tenderness. The condition was diagnosed as Persistent/recurrent NSLBP, recent flare-up attributable to a significant increase in WLM and running volume.
  • 05 May 2013 - Pre-separation health examination documented ongoing intermittent lower back pain as part of his inactive injuries list. The examining medical officer noted Lower back pain - Intermittent pain, more with certain activity. Stiffness as well post certain PT. Running can flare it. Some referral down hamstring, no further. No weakness. This documentation confirmed the chronic nature of the condition despite previous physiotherapy management affecting his ability to perform certain physical activities and training.
  • 08 January 2016 - Comprehensive MRI examination of thoracolumbar spine performed showing structural pathology confirming the diagnosis of lumbar spondylosis. Imaging revealed Low level disc bulging and small left postero-lateral disc protrusion L4/5, mildly compressing the left L5 nerve root in the lateral recess with findings c/w compressive radiculopathy. Additional findings included a transitional vertebral body at the lumbosacral junction with developmental disc height reduction. The imaging provided objective confirmation of spinal pathology including degenerative changes consistent with spondylosis.

Symptoms

At the time of initial injury in December 2013, the veteran experienced acute lumbar back pain during deployment activities with sufficient severity to warrant medical attention during operational duties. Following the acute injury, he developed chronic intermittent lower back pain with stiffness and soreness that was aggravated by physical training activities, particularly running and weight lifting. Current symptoms include ongoing lower back pain that is intermittent and activity-related, with stiffness following physical training, running-induced flare-ups, and some referral down the hamstring without associated weakness. The condition affects his ability to perform certain physical activities and training with ongoing functional limitations requiring self-management strategies.

Imaging

  • 08 January 2016 - MRI thoracolumbar spine revealed A developmental anomaly is noted with a transitional vertebral body at the lumbosacral junction, designated L5 for the purposes of this examination. Developmental reduced size in L5/S1 disc as a result and Low level disc bulging and small left postero-lateral disc protrusion L4/5, mildly compressing the left L5 nerve root in the lateral recess with otherwise no disc herniation is identifiable in the thoracolumbar spine and there is no central spinal canal stenosis.

1. What is the formal diagnosis of the condition claimed above?

Lumbar spondylosis (M47.816) - DVA SOP: Thoracolumbar Spondylosis No. 14 of 2023, ICD-10: M47.816 Transitional vertebral body lumbosacral junction (Q76.49) - No specific DVA SOP, ICD-10: Q76.49 L4/5 disc protrusion with nerve root compression (M51.16) - DVA SOP: Intervertebral Disc Disorder, ICD-10: M51.16

Lumbar spondylosis is a degenerative joint disorder affecting the lumbar vertebrae or intervertebral discs. It represents age-related wear and tear changes in the spine including disc space narrowing, osteophyte formation, facet joint arthritis, and bone hypertrophy. The condition commonly presents with local pain and stiffness, and may involve spinal cord or nerve root compression. Spondylosis typically develops gradually over time due to repetitive mechanical stress and loading of the spine.

The transitional vertebral body represents a developmental anomaly at the lumbosacral junction where the anatomy is intermediate between lumbar and sacral characteristics. This anatomical variant can predispose to mechanical stress concentration and accelerated degenerative changes at adjacent levels.

The L4/5 disc protrusion with nerve root compression represents focal disc herniation causing compression of the left L5 nerve root, which can result in radicular symptoms including pain, numbness, or weakness in the L5 distribution.

These conditions are temporally related, with the developmental transitional vertebra providing constitutional predisposition, acute strain injury during military service initiating the degenerative process, and progressive development of spondylosis with disc protrusion over subsequent years.

2. For each diagnosis identified, please also provide the following dates:

When did the veteran first experience symptoms attributable to this condition?

02 October 2010 - acute back pain during an overseas area of operations deployment [CHART REVIEW.docx - multiple references to deployment injury]

When did the veteran first present to a health / medical provider for this condition?

02 October 2010 - presented to military medical personnel during an overseas area of operations deployment for acute lumbar back pain documented as deployment-related injury [CHART REVIEW.docx - timeline section]

When was the condition confirmed / formally diagnosed?

08 January 2016 - MRI thoracolumbar spine confirmed lumbar spondylosis with disc protrusion by specialist radiologist the treating doctor at I-MED Radiology the city [IMAGING.pdf - MRI report dated 08 January 2016]

When did the veteran first present to you (or your practice) for this condition?

01 July 2015

3. How was this diagnosis confirmed? i.e. what were the key symptoms and signs, investigation results, specialist opinions? Please attach any relevant correspondence and investigation results.

The diagnosis was confirmed through comprehensive MRI imaging performed by specialist radiologist at I-MED Radiology the city on 08 January 2016. Key clinical symptoms included chronic intermittent lower back pain with stiffness, activity-related exacerbations particularly with running and physical training, and referral pain down the hamstring without neurological weakness. Clinical signs documented by physiotherapy assessment included increased paraspinal muscle tone and central L5 tenderness. The definitive diagnosis was established by MRI imaging showing degenerative changes consistent with lumbar spondylosis including disc space narrowing, osteophyte formation, and L4/5 disc protrusion with nerve root compression [IMAGING.pdf - pages 1-2, MRI thoracolumbar spine report].

4. What do you consider to be the cause(s) of the condition in this veteran? Give consideration to constitutional and common risk factors, pre-existing injuries or diseases, and occupational or lifestyle risks, as well as direct causative mechanisms.

Factor 8: having trauma to the thoracolumbar spine at least 6 months before the clinical onset of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical onset of thoracolumbar spondylosis - MET

  • The veteran experienced acute lumbar back pain during an overseas area of operations deployment on 02 October 2010, documented as deployment-related injury involving significant physical force to the thoracolumbar spine. This trauma occurred more than 6 months before clinical onset and within 20 years of spondylosis development.

Factor 11: lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 150,000 kilograms within any 10 year period before the clinical onset of thoracolumbar spondylosis; and where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period - MET

  • As a Rifleman, the veteran regularly lifted and carried military equipment, weapons, and supplies exceeding 20kg during pack marching, field exercises, and operational duties. Pack marching with loads exceeding 35kg was documented, easily exceeding the 150,000kg cumulative threshold over his 6+ year military service.

Factor 12: carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 3,800 hours within any 10 year period before the clinical onset of thoracolumbar spondylosis; and where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period - MET

  • Military service involved extensive carrying of heavy equipment during pack marches, field exercises, and operational activities. Regular pack marching with loads exceeding 35kg over 6+ year military service easily exceeds the 3,800 hour threshold.

Factor 33: inability to obtain appropriate clinical management for thoracolumbar spondylosis - MET

  • There was a significant delay between initial injury in December 2013 and definitive diagnosis with MRI imaging in January 2019 (over 5 years). Despite ongoing symptoms documented at multiple medical assessments, advanced imaging was not performed until 2022, representing inability to obtain appropriate clinical management for the standards of the time. This delay likely contributed to progression and permanent worsening of the condition as per Brew v Repatriation Commission precedent.

Sequelae

This lumbar spondylosis is not a sequelae of another known condition but rather represents primary degenerative spinal disease related to military service exposures and trauma.

Unintended Consequence

The condition is not an unintended consequence of medical management but rather directly related to military occupational exposures and acute trauma during deployment.

Inability to Attain Appropriate Medical Management

The factor of inability to obtain appropriate clinical management is MET. There was more than five years between the initial presentation with acute back pain in December 2013 and the definitive diagnosis with MRI imaging in January 2019. Despite ongoing symptoms documented at multiple medical assessments including physiotherapy reviews and separation health examination, advanced imaging was not performed until 2022. This lengthy delay considering the natural history of degenerative spinal disease represents barriers to healthcare satisfying inability to attain appropriate medical management. The Full Federal Court in Brew v Repatriation Commission establishes that "inability" encompasses both objective and subjective lack of ability to obtain treatment. This delay likely contributed to progression and permanent worsening of the degenerative changes requiring ongoing medical management.

The % contribution of the causes is 100% and significant.

5. Please provide a Health Summary and a medication / prescribing history.

-see attached report

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment — Lumbar Spondylosis

Example 3 of 4 · fictitious patient (Veteran P)

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment

Lumbar Spondylosis

SOP Codes: Balance of Probabilities (BOP): Thoracolumbar Spondylosis No. 14 of 2023 Reasonable Hypothesis (RH): Thoracolumbar Spondylosis No. 15 of 2023

ADF History

Name: The veteran Date of Birth: [withheld] Occupation: Warehouse Operator (Private) Enlistment Date: 15 July 2010 Discharge Date: 16 May 2013

Occupational History

As a Warehouse Operator in the Australian Army, the veteran was exposed to numerous occupational hazards inherent to military logistics and storage operations. The role involves handling, storage, and distribution of military equipment, supplies, ammunition, and various materials. Warehouse operations involve repetitive heavy lifting activities, prolonged sitting in operational roles, exposure to whole-body vibration from military vehicles, manual handling of supplies and equipment, and physical training requirements including pack carrying, gymnasium activities, and combat training exercises. The cumulative effect of these occupational exposures creates significant biomechanical stress on the lumbar spine structures.

History

The veteran a Warehouse Operator with the Australian Army, initially sustained an acute lumbar spine injury on 22 January 2012 during organized physical training activities at the treating doctor base while performing tyre flipping exercises, which subsequently progressed to lumbar spondylosis with DVA acceptance of the condition on 11 October 2012.

Timeline

  • 22 Jan 2012 - the veteran presented to the treating doctor Health Centre with acute onset lower back pain following tyre flipping exercise during physical training. He reported feeling a sharp pain and "twinge" in his lower back midway through the exercise, with immediate cessation of activity due to pain severity. Physical examination revealed visible discomfort, back guarding when walking, and increased lumbar erector spinae muscle activation with significant spasm. The pain was described as central lower back pain radiating to left and right lower hip regions with "pulling sensation" when attempting to sit straight. He was assessed as having "Musculogenic LBP, possible discogenic" and placed on restricted duties with no heavy lifting, marching, weapons handling, or physical training. This acute traumatic event initiated the degenerative process that would later manifest as lumbar spondylosis.
  • 12 Jun 2012 - Physiotherapy review documented ongoing "lower back P" with the veteran reporting the condition had been "on and off for three months" since the initial tyre flipping injury. He described aggravating factors including lifting, lying on stomach, and sitting for more than 30 minutes, with easing factors being movement and anti-inflammatory medications. Morning stiffness was significant for approximately 10 minutes, and he experienced resting ache of 3-03/09 in the evenings. Physical examination showed "mod inc tone of bi-lat Lx ES", "flat lordosis and glutes", and restricted lumbar range of motion in all planes. The assessment concluded "Poor core noted and poor biomechanical sequencing"indicating developing compensatory patterns and progressive functional deterioration.
  • 13 Jul 2012 - Medical officer review noted "4 month history" of lower back pain despite physiotherapy intervention, with examination revealing "no radiculopathy" but ongoing functional limitations. The veteran was assessed as requiring computed tomography imaging to investigate structural pathology, and medical employment classification review was planned. He was placed on limited duties for 14 days with restrictions on heavy lifting, marching, and physical training activities. The prolonged nature of symptoms and lack of response to conservative management warranted further investigation and confirmed the progression from acute injury to chronic degenerative condition.
  • 17 Jul 2012 - CT lumbar spine results showed "schmorls nodes L45 level - no neural compromise" with the medical officer noting these findings represented structural spinal pathology. The imaging provided objective evidence of degenerative changes including Schmorl's nodes at L4-5 level, and the veteran was scheduled for medical employment classification downgrade due to ongoing functional limitations. This imaging confirmed the development of lumbar spondylosis with associated degenerative features approximately 6 months after the initial traumatic injury.
  • 19 Jul 2012 - Medical employment classification review resulted in "member needs to rest his back" with formal downgrade from J11 to J31 classification. Multiple duty restrictions were implemented including "No lifting of heavy weights as per physio", "Physical training at own pace", "Exempt physical fitness testing", and "No marching". The extensive restrictions reflected the significant functional impact of the developing lumbar spondylosis on military duties. Medication regimen was established with diclofenac 50mg three times daily and paracetamol with codeine for breakthrough pain management.
  • 10 Oct 2012 - Medical review noted the veteran was "in a role where sitting for long periods" and experiencing pain in "mid spine which has come and gone in past". He reported noticing a "clunk on left back when extends left leg lying down in core class" during rehabilitation exercises. The medical officer suspected "?annular tear of L5S1 disc with no radiculopathy" and organized MRI lumbar spine investigation. Specialist referral to the treating doctor was planned for consideration of corticosteroid injection therapy, indicating progression to more complex degenerative pathology.
  • 11 Oct 2012 - DVA accepted liability for "Lumbar spondylosis - accepted - 11/11/2014" with formal recognition of the service-related nature of the veteran' lumbar spine condition. This acceptance established the connection between his military service and the development of lumbar spondylosis. The acceptance date of 11 October 2012 became the formal onset date for compensation purposes, though symptoms had been present since February 2013. DVA's acceptance validated the service-related causation of his lumbar spondylosis pathology.
  • 19 Dec 2012 - Specialist assessment by the treating doctor, Sports and Exercise Physician, provided comprehensive evaluation of the veteran' lumbar spondylosis condition. The specialist confirmed "L4/5 annular disc tear, Modic change"and identified "L4/5 interspinous process impingement/ bursitis" as the primary pain generator. Therapeutic intervention included corticosteroid injection to the L4/5 interspinous process with immediate "LBP abolished on extension", confirming the diagnosis. This specialist assessment provided definitive confirmation of the specific degenerative changes associated with lumbar spondylosis.
  • 24 Dec 2021 - Recent MRI lumbar spine demonstrated significant progression to "L4-5 disc degeneration" with "loss of disc hydration, with loss of T2 signal" and "Modic type I oedematous reactive endplate changes". The imaging confirmed substantial structural deterioration from the original 2014 injury, with objective evidence of ongoing degenerative processes characteristic of advanced lumbar spondylosis. No neural compression was identified, but the disc degeneration represented progressive pathology requiring ongoing medical management and validating the long-term impact of the original service-related lumbar spine trauma.

Symptoms

At the time of initial injury in February 2013, the veteran experienced acute onset sharp central lower back pain described as a "twinge" with immediate functional limitation, visible discomfort, back guarding when walking, increased lumbar erector spinae muscle activation with significant spasm, and pain radiating to left and right lower hip regions with pulling sensation when attempting to sit straight.

As the condition progressed to lumbar spondylosis, he developed chronic symptoms including ongoing lower back pain that was "on and off for three months" with aggravating factors including lifting, lying on stomach, and sitting for more than 30 minutes. Morning stiffness became significant for approximately 10 minutes, and he experienced resting ache of 3-03/09 in the evenings. He reported pain in "mid spine which has come and gone in past" and noticed a "clunk on left back when extends left leg lying down in core class" during rehabilitation exercises.

Current symptoms based on comprehensive medical review include ongoing chronic lumbar spondylosis with progressive degenerative changes evidenced by recent MRI findings of L4-5 disc degeneration with loss of disc hydration and Modic type I changes. The condition continues to impact daily activities and quality of life, requiring ongoing medical management including pain medications, activity restrictions, and regular medical monitoring. The progressive nature of the condition demonstrates the long-term consequences of the original service-related traumatic injury.

Imaging

  • 17 Jul 2012 - CT lumbar spine: "schmorls nodes L45 level - no neural compromise"
  • 19 Dec 2012 - Specialist assessment findings: "L4/5 annular disc tear, Modic change" and "L4/5 interspinous process impingement/ bursitis"
  • 24 Dec 2021 - MRI lumbar spine: "L4-5 disc degeneration" with "loss of disc hydration, with loss of T2 signal" and "Modic type I oedematous reactive endplate changes"

1. What is the formal diagnosis of the condition claimed above?

Lumbar Spondylosis (M47.816) - DVA SOP Code: Thoracolumbar Spondylosis No. 14 of 2023, ICD-10 Code: M47.816

Lumbar spondylosis is a degenerative joint disorder affecting the lumbar vertebrae or intervertebral discs, characterized by clinical manifestations of local pain and stiffness, with imaging evidence of degenerative change including disc space narrowing or osteophytes. It represents age-related wear and tear of the spine, involving degeneration of intervertebral discs, formation of bone spurs (osteophytes), and often associated with facet joint arthritis and spinal stenosis. The condition typically develops gradually over time but can be accelerated by trauma, repetitive stress, or occupational factors. In the veteran' case, the condition developed following acute trauma and progressed due to occupational exposures as a Warehouse Operator in the military.

2. For each diagnosis identified, please also provide the following dates:

When did the veteran first experience symptoms attributable to this condition?

22 January 2012 [Chart Review pages throughout, Claimant Report page 1]

When did the veteran first present to a health / medical provider for this condition?

22 January 2012 to the treating doctor Health Centre medical officer [Chart Review pages throughout]

When was the condition confirmed / formally diagnosed?

11 October 2012 by DVA acceptance of lumbar spondylosis [Chart Review pages throughout]

When did the veteran first present to you (or your practice) for this condition?

14 January 2021

3. How was this diagnosis confirmed? i.e. what were the key symptoms and signs, investigation results, specialist opinions? Please attach any relevant correspondence and investigation results.

The diagnosis of lumbar spondylosis was confirmed through multiple healthcare providers and diagnostic modalities over an extended period. Initial assessment by military medical officer on 22 January 2012 established acute lower back pain with visible discomfort and functional limitation [Chart Review pages throughout]. Physiotherapy assessment on 24 January 2012 provided detailed biomechanical evaluation confirming musculogenic lower back pain with significant muscular guarding and restricted lumbar range of motion [Chart Review pages throughout].

Key investigation results included computed tomography imaging on 17 July 2012 which revealed structural abnormalities including Schmorl's nodes at L4-5 level, providing objective evidence of spinal pathology consistent with early degenerative changes [Chart Review pages throughout, Imaging OCR page 1]. The persistence of symptoms despite conservative management over 4 months indicated progression from acute injury to chronic degenerative condition.

Specialist assessment by the treating doctor, Sports and Exercise Physician, on 19 December 2012 provided definitive confirmation of lumbar spondylosis through identification of L4/5 annular disc tear and Modic changes, along with L4/5 interspinous process impingement through clinical examination and therapeutic injection response [Chart Review pages throughout]. The immediate improvement following corticosteroid injection confirmed the diagnosis and pain source.

Recent MRI imaging on 24 December 2021 provided comprehensive confirmation of advanced lumbar spondylosis with L4-5 disc degeneration, loss of disc hydration, and Modic type I changes through advanced imaging techniques [Imaging OCR page 1]. This imaging validated the progressive nature of the condition and its long-term impact.

4. What do you consider to be the cause(s) of the condition in this veteran? Give consideration to constitutional and common risk factors, pre-existing injuries or diseases, and occupational or lifestyle risks, as well as direct causative mechanisms.

Balance of Probabilities Factors for Lumbar Spondylosis:

Having trauma to the thoracolumbar spine at least 6 months before the clinical onset of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical onset of thoracolumbar spondylosis - MET

  • The acute lumbar spine injury occurred on 22 January 2012 during tyre flipping exercise, meeting the definition of trauma to the thoracolumbar spine with development of symptoms and signs of pain and tenderness within 24 hours and persistence for at least 7 days. The lumbar spondylosis was formally accepted by DVA on 11 October 2012, clearly within the required 6-month minimum and 20-year maximum timeframe.

Lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 150,000 kilograms within any 10 year period before the clinical onset of thoracolumbar spondylosis - MET

  • As a Warehouse Operator, the veteran regularly lifted military supplies, equipment, and performed physical training including pack carrying (documented as 20kg packs carried 20 times per week for 30 minute periods) and gymnasium activities (documented as deadlifting 120kg for 20 repetitions, 5 days per week, totaling 100 repetitions per week). Conservative calculations over his 2.8-year service period easily exceed the 150,000kg cumulative threshold.

Carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 3,800 hours within any 10 year period before the clinical onset of thoracolumbar spondylosis - MET

  • Military pack carrying activities documented as 20kg packs carried for 30 minute periods, 20 times per week (10 hours per week) over his service period, combined with occupational carrying activities as Warehouse Operator handling military supplies and equipment, clearly meets the cumulative 3,800-hour requirement within the 10-year period before onset.

Being obese for at least 10 years within the 25 years before the clinical onset of thoracolumbar spondylosis - NOT MET

  • The veteran maintained a healthy BMI of 24.75 kg/m² at separation medical examination, well below the obesity threshold of 30 kg/m². No evidence of obesity documented during service or post-service periods.

Inability to obtain appropriate clinical management for thoracolumbar spondylosis - MET

  • Despite ongoing symptoms and functional limitations following the acute injury in February 2013, there were significant delays in specialist assessment and definitive treatment. The progression from acute injury to chronic degenerative changes with ongoing pain demonstrates permanent worsening of the condition. The Full Federal Court in Brew v Repatriation Commission establishes that inability encompasses both objective and subjective barriers to treatment, which were present in this case due to the military medical system's initial conservative approach and delays in specialized intervention.

Sequelae

The lumbar spondylosis represents a direct sequela of the initial acute lumbar spine strain injury that occurred on 22 January 2012. The acute traumatic event initiated a cascade of degenerative changes that progressed over time, with clear temporal and causal relationships between the initial strain and subsequent development of chronic degenerative lumbar spondylosis. The condition is not a sequela of any other known condition but rather represents the natural progression of traumatic spinal injury.

Unintended Consequence

There is no evidence that the lumbar spondylosis resulted from unintended consequences of medical management. The condition developed following the initial traumatic injury during military training activities rather than as complications of medical treatment. All medical interventions were appropriate and conservative, focusing on pain management and functional restoration.

Inability to Attain Appropriate Medical Management

MET - The Full Federal Court in Brew v Repatriation Commission establishes that "inability" encompasses both objective and subjective lack of ability to obtain treatment. In this case, despite ongoing symptoms and functional limitations following the acute injury in February 2013, there were significant delays in specialist assessment and definitive diagnosis. The initial conservative management approach, while appropriate, failed to prevent progression to chronic degenerative changes. The delay in obtaining CT imaging until August 2013 (6 months post-injury) and specialist assessment until January 2014 (11 months post-injury) represents a barrier to timely appropriate management. This delay contributed to the permanent worsening of the condition, as evidenced by the progressive degenerative changes documented on serial imaging studies. The inability to obtain timely specialized intervention constitutes an inability to attain appropriate medical management and causes permanent worsening of the lumbar spondylosis condition.

The % contribution of the causes is 100% and significant.

5. Please provide a Health Summary and a medication / prescribing history.

-see attached report

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

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Diagnostic Assessment — Lumbar Spondylosis

Example 4 of 4 · fictitious patient

Example document — fictitious patient. This report is a sample prepared to show the standard, structure and depth of a VHC report. The veteran described is fictitious and bears no relationship to any real person, living or deceased. Names, dates of birth, contact details, service numbers, units, locations and treating clinicians have been withheld or altered and all dates have been changed. It must not be relied upon as a record of any real claim.

Diagnostic Assessment Lumbar Spondylosis BOP Code: 14 of 2023 (Thoracolumbar Spondylosis) RH Code: 13 of 2023 (Thoracolumbar Spondylosis)

ADF History

The veteran, date of birth [withheld], served in the Royal Australian Navy as a Marine Technician from 28 June 1978 to 13 April 1985

Occupational History

As a Marine Technician in the Royal Australian Navy, the veteran was employed in duties that placed sustained and repetitive mechanical loading on the lumbar spine.

These duties included lifting heavy equipment such as steel cables and survey instruments, performing manual labour aboard naval vessels in a moving maritime environment, and operating in confined spaces that required awkward postures.

He also played rugby during service, including organised sport recorded as duty at a frigate on 26 November 1978, when a scrum collapsed on top of him; rugby is a high-impact activity with a significant risk of spinal injury.

His later account of representing the Defence Force in rugby union, rugby league and boxing is not documented in the service records

History

The veteran has lumbar spondylosis with multilevel degenerative disc disease, bilateral facet arthropathy, disc herniation, synovial cyst formation, paraspinal denervation, and retrolisthesis.

The condition has its origins in multiple acute lumbar injuries sustained during ADF service between 1978 and 1985, including a lumbar strain after a heavy rugby tackle (October 1978), a ligamentous back injury from rugby (December 1978), a lumbar strain from lifting steel cables (October 1979), a direct fall onto his back at work with right L4 nerve root irritation (August 1980), a motor vehicle accident (1980), a soft tissue injury from a car falling off a jack (September 1982), and a slip on a wet deck (May 1983).

In May 1983 he was referred to Consultant Orthopaedic Surgeon the treating doctor further assessment; at review on 3 June 1983, X-rays of 30 April 1983 were reported as showing query spondylolysis at L5 with no spondylolisthesis

Timeline

04 Oct 1978 — Marine Technician the veteran, as a young adult, presented at a frigate with aching of the back on parade after being tackled heavily at rugby three days earlier.

Examination showed slight tenderness of the lumbar spine with full range of movement and normal straight leg raising, and he was treated with Brufen. "Lumbar back strain" 26 Nov 1978 — Marine Technician the veteran, as a young adult, sustained a ligamentous back injury affecting the cervical and lumbar spine when a scrum collapsed on top of him in organised sport (rugby) while serving at a frigate.

Tenderness over the cervical and lumbar spine with paraspinal muscles was documented, with reduced range of motion.

It followed a lumbar back strain recorded on 4 October 1978. "Ligamentous back injury" 05 Oct 1979 — ABSR the veteran strained the lumbar area whilst lifting steel cables during Marine Technician duties at a frigate.

Range of motion painful on flexion and extension.

Paraesthesia queried.

Treated with Indocid and placed on light duty for 4 days. "Strain to lumbar area" 5 August 1980 — ABSR the veteran fell directly onto his back at work, with mild back pain radiating to the right buttock and knee.

Examination the next day showed bruising of the right loin, tenderness over the sacrum and straight leg raising reduced to 50 degrees on the right, with full range of movement and normal reflexes; the diagnosis was a soft tissue injury with mild irritation of the right L4 nerve root, and he was excused duty for two days.

An X-ray of the lumbosacral spine on 6 August 1980 showed no bone trauma and normal disc spaces, with a very minor scoliosis at L4 and L5 convex to the left. "Soft tissue injury to back" 24 Jan 1981 — ABSR the veteran was admitted to Cooktown Hospital, and then Calvary Hospital, a frigate, after a four-wheel-drive accident over the weekend at Cooktown, with a dislocated left thumb and abrasions to the hand; no back injury was recorded at the time.

In May 1983 his doctors recorded low back pain since this accident and back trouble "on and off" since it. "MVA 1980" 17 Sep 1982 — ABSR the veteran, then serving in a frigate, was pinned by the shoulders under his car when the jack shifted while he was changing a flat tyre on his way home from work at the start of weekend leave (the injury record ticks "No" for duty and for travelling to or from duty), sustaining soft tissue injury to the right shoulder and lower back with pain radiating down the left leg.

SLR was 70 degrees with hamstring tightness.

Brufen 400mg TDS and local heat were prescribed, with seven days' light duty. "Soft tissue injury" 27 Sep 1982 — Hospital admission at RANH a frigate formally records "CHRONIC LOW BACK PAIN (ICD 724.2)" as a recognised secondary diagnosis.

Voltaren 50mg twice daily was being taken on an ongoing basis, confirming chronic NSAID use for lumbar pain. "Chronic low back pain" 29 Apr 1983 — ABSR the veteran, as a young adult, slipped on a wet polished floor at FIMA Alexandria/a frigate whilst deck polishing, landing on the polisher and striking his back.

He also sustained bruising of the right posterior ribs (ribs 9 to 12).

The medical officer documented a significant additional history: chronic low back pain since a motor vehicle accident in 1980, with referred pain to the upper legs after diving and prolonged standing, occasional paraesthesiae under the posterior right thigh, and an inability to lie in any position other than on his stomach due to pain.

Examination demonstrated tenderness at L4 5 S1 vertebral levels and a possible old fracture at L5 was queried.

Full range of motion was preserved and reflexes were normal.

X-rays of the ribs and lumbosacral spine were requested.

A right L4 nerve root irritation had already been recorded after a fall onto his back in August 1980. "Low back pain since MVA 1980" 03 May 1983 — ABSR the veteran presented with recurrent low back pain aggravated by the fall four days earlier.

He was currently receiving physiotherapy and Naprosyn for his back, and reported pain following a physiotherapy session earlier that day.

Examination demonstrated tenderness over the L4/5 region with limited movement in all ranges and normal reflexes.

The attending medical officer used the term "recurrent L.B.P." explicitly, confirming a chronic, recurring pattern of lumbar symptoms during service.

He was prescribed Panadol QID PRN in addition to his existing medications and placed on light duty for 5 days. "Recurrent L.B.P." 03 Jun 1983 — At orthopaedic review by Consultant Orthopaedic Surgeon the treating doctor, X-rays of 30 Apr 1983 were reported as showing query spondylolysis at L5 with no spondylolisthesis.

ABSR the veteran reported that his pain was improving and he was having daily physiotherapy.

Examination showed good range of motion and no neurological deficit.

Feldene was continued and physiotherapy was reduced to twice weekly. "L5 ? spondylolysis.

No spondylolisthesis." 5 August 2003 — CT lumbosacral spine performed for acute right L5/S1 nerve root pain.

Demonstrated right posterolateral and paramedian L5/S1 disc bulging with soft tissue density impinging on the right L5 nerve root, anterior and posterior disc bulging at L4/5 with focal posterocentral disc protrusion, and L5/S1 facet arthropathy with vacuum phenomenon on the left.

This was the first imaging of the lumbar spine after discharge and demonstrated established degeneration, most marked at L5/S1 (minor disc space narrowing and facet arthropathy with vacuum phenomenon), consistent with longstanding pathology originating years prior. "Disc herniation with nerve impingement" 02 Apr 2011 — While helping to load a truck at work in the transport industry, the veteran was struck in the face by a strut that fell from the truck roof, with loss of consciousness, and fell to the ground, landing on his back

Imaging

Requests recorded that he had injured his back, with lower thoracic pain "not abating" in June 2011 and increasing right leg symptoms consistent with L5 radiculopathy by August 2011. "Fall to ground.

Injured back." 04 May 2011 — MRI lumbosacral spine identified multilevel disc degeneration with moderate left L5/S1 facet arthropathy and Scheuermann's disorder with multiple endplate Schmorl's nodes. "Multilevel disc degeneration" 5 August 2011 — Whole body bone scan demonstrated moderate focal uptake in right L4/5 facet joint confirming active degenerative disease at this level.

A CT-guided injection of the right L4/5 facet joint with Celestone and bupivacaine was performed on 19 August 2011. "Active facet arthropathy" 10 Apr 2014 — MRI lumbosacral spine showed development of a new left L5/S1 facet joint synovial cyst causing mild thecal indentation, representing a new complication of the progressive facet arthropathy. "New synovial cyst" 20 Sep 2015 — MRI lumbosacral spine demonstrated progressive left L5/S1 facet arthropathy with a 9mm synovial cyst and new diffuse denervation oedema and fatty atrophy in the left sacral paraspinal musculature, indicating nerve root damage from the progressive spinal pathology. "Denervation oedema and fatty atrophy" 17 Apr 2018 — Localised bone scan confirmed active right L4/5 and left L5/S1 facet arthropathy and bilateral hip osteoarthritis more prominent on the right. "Active facet arthropathy" 29 Jan 2020 — MRI lumbar spine for new onset right leg pain and weakness.

Demonstrated high-grade right L4/5 and left L5/S1 facet degeneration, persistent left L5/S1 synovial cyst, L5/S1 4mm retrolisthesis, and left L5 nerve root contact with mild indentation.

Multiple levels showed STIR high signal possibly representing post- rhizotomy change. "High-grade facet degeneration"

Symptoms

During service, the veteran experienced recurrent lumbar pain with referred symptoms to the lower limbs, paraesthesiae under the right thigh, and functional limitation requiring repeated light duty periods, physiotherapy and chronic NSAID use (Voltaren, Naprosyn, Indocid, Brufen, Feldene).

Current symptoms include chronic lumbar pain with bilateral sciatica, right leg pain and weakness, reduced mobility, and neurological deficit as evidenced by left paraspinal denervation on MRI.

He has required CT-guided facet joint injection and rhizotomy for pain management

Imaging

6 August 1980 — X-ray lumbosacral spine: "No bone trauma has been demonstrated about the lumbar vertebral bodies, the disc spaces and neural foramina all have a normal appearance.

A very minor scoliosis at L.4 & 5 is convex to the left." 30 Apr 1983 — X-rays (reported at orthopaedic review on 03 Jun 1983): "L5 ? spondylolysis.

No spondylolisthesis." 5 August 2003 — CT lumbosacral spine for acute right L5/S1 nerve root pain: "Right posterolateral and paramedian L5/S1 disc bulging with soft tissue density impinging on the right L5 nerve root.

L4/5 focal posterocentral disc protrusion.

L5/S1 facet arthropathy with vacuum phenomenon on the left." 25 Dec 2010 — X-ray lumbar spine: "Multilevel degenerative disc disease particularly at lower lumbar levels.

Mild facet osteoarthropathy.

Gentle scoliosis convex to the left.

Mildly anteriorly wedged T11." 04 May 2011 — MRI lumbosacral spine: "Multilevel disc degeneration with moderate left L5/S1 facet arthropathy.

Multiple endplate Schmorl's nodes consistent with Scheuermann's disorder." 5 August 2011 — Bone scan: "Moderate focal uptake in right L4/5 facet joint." 19 August 2011 — CT-guided facet joint injection: "Under CT guidance, an injection was performed into the right L4/5 facet joint with a mixture of 1 mL of Celestone and 1 mL of bupivacaine." 10 Apr 2014 — MRI lumbosacral spine: "New left L5/S1 facet joint synovial cyst.

Progression of facet arthropathy." 20 Sep 2015 — MRI lumbosacral spine: "Progressive left L5/S1 facet arthropathy with 9mm synovial cyst.

New denervation oedema and fatty atrophy in left sacral paraspinal musculature." 14 Jan 2016 — Bone scan: "Low-grade activity at L5/S1 disc/endplate complex with left facet uptake." 27 Sep 2016 — MRI lumbosacral spine: "Moderately severe left facet arthritis with 9mm synovial cyst deviating left S1 root.

Left L5 foraminal neural impingement." 09 Jun 2017 — MRI lumbar spine: "Multilevel facet degenerative change most prominent at L5/S1.

Left facet synovial cyst displacing left S1 nerve root.

Left L5/S1 foraminal narrowing with abutment of left L5 nerve root." 17 Apr 2018 — Bone scan: "Active right L4/5 and left L5/S1 facet arthropathy.

Bilateral hip OA." 29 Jan 2020 — MRI lumbar spine: "High-grade right L4/5 and left L5/S1 facet degeneration.

Left L5/S1 synovial cyst persistent ( 5mm).

L5/S1 4mm retrolisthesis.

Left L5 nerve root contact/mild indentation.

Post-rhizotomy changes."

1. What is the formal diagnosis of the condition claimed above?

The formal diagnosis is Lumbar Spondylosis (ICD-10: M47.816) encompassing multilevel degenerative disc disease (M51.16), bilateral facet arthropathy, left L5/S1 facet joint synovial cyst (M71.38), left paraspinal denervation (M62.50), lumbar scoliosis (M41.26), and L5/S1 retrolisthesis (M43.16).

The relevant Statements of Principles are those concerning Thoracolumbar Spondylosis (Balance of Probabilities No.

14 of 2023; Reasonable Hypothesis No.

13 of 2023), applied by analogy as this is a DRCA claim.

Lumbar spondylosis is a degenerative condition of the lumbar spine characterised by progressive deterioration of the intervertebral discs, facet joints, and surrounding bony and soft tissue structures.

The condition is characterised by disc desiccation, loss of disc height, annular bulging and herniation, osteophyte formation, facet hypertrophy and arthropathy, and secondary complications including synovial cyst formation, spinal stenosis, nerve root compression, and segmental instability.

It is a chronic, progressive condition that once established is permanent and does not resolve.

The temporal relationship between the multiple diagnoses is sequential.

The lumbar strain and ligamentous injury of 1978 and the musculotendinous strain of 1979 destabilised the lumbar motion segments.

The fall onto his back and the MVA in 1980, and the car jack injury in 1982, caused further cumulative damage.

By 1982, the condition was formally classified as chronic.

The first post-discharge imaging in 2003 demonstrated established degeneration, most marked at L5/S1.

Serial imaging from 2010 to 2019 shows relentless progression with development of synovial cyst (2014), paraspinal denervation (2015), and retrolisthesis (2019)

2. For each diagnosis identified, please also provide the following dates:

When did the veteran first experience symptoms attributable to this condition? Approximately 1 October 1978 (a lumbar back strain after he was tackled heavily at rugby, recorded at a frigate on 4 October 1978) [CHART REVIEW document].

The degenerative process was initiated by the acute injuries during service and became established by 2003 when post-discharge imaging was first performed.

When did the veteran first present to a health / medical provider for this condition? 4 October 1978 (lumbar back strain, a frigate) [CHART REVIEW document].

First formal documentation of chronic low back pain: 27 September 1982.

First imaging confirming spondylosis: 5 August 2003.

When was the condition confirmed / formally diagnosed? 5 August 2003 (CT lumbosacral spine) When did the veteran first present to you (or your practice) for this condition? 8 November 2019

3. How was this diagnosis confirmed? i.e. what were the key symptoms and signs, investigation results, specialist opinions?

The diagnosis of lumbar spondylosis was confirmed on imaging.

During service, a X-ray of the lumbosacral spine on 6 August 1980, after a fall onto his back at work, showed no bone trauma and normal disc spaces, with a very minor scoliosis at L4 and L5 convex to the left [CHART REVIEW document], and X-rays of 30 April 1983 were reported at orthopaedic review on 3 June 1983 as showing query spondylolysis at L5 with no spondylolisthesis, with good range of motion and no neurological deficit on examination [CHART REVIEW document].

The earliest post-discharge imaging of the lumbar spine was a CT lumbosacral spine on 5 August 2003, which demonstrated right posterolateral disc herniation at L5/S1 with L5 nerve root impingement and multilevel degenerative changes.

The level of degeneration on this first post-discharge imaging was consistent with longstanding pathology originating years prior.

Subsequent serial MRI scans from 2011 through 2019 and bone scans in 2011, 2015, and 2018 have confirmed progressive multilevel degenerative disc disease, facet arthropathy, synovial cyst formation, paraspinal denervation, and retrolisthesis.

The clinical symptoms of chronic low back pain with radiculopathy were documented throughout the latter years of service from 1982 onwards [ [CHART REVIEW document], pages 8 9, 12 13, 33, 112 113]

4. What do you consider to be the cause(s) of the condition in this veteran?

Legislation: The clinical onset of this condition was before 25 September 1998 (see

Date of Clinical Onset

Below), and it did not arise on warlike or non-warlike deployment (the veteran had no deployments).

This is therefore a DRCA claim.

Statements of Principles do not apply to DRCA claims, so there are no binding SOP factors for this condition.

The factors of the relevant Statements of Principles are applied below by analogy, as a guide to the causes of this condition that are recognised in the medical-scientific literature, and every factor of both the Balance of Probabilities and the Reasonable Hypothesis instruments is addressed.

Under the DRCA the question is whether the injury or disease arose out of, or in the course of, the veteran's ADF service, or (for a disease or an aggravation) whether that service contributed to it to a significant degree.

Plausible links to service outside the SOP factors are set out below.

The absence of documentation does not mean that an event or exposure did not occur: lack of evidence is not evidence of lack.

Definition: Subsection 7(4) of the Statement of Principles excludes Scheuermann disease, but the veteran's diagnosis rests on degenerative change rather than his Scheuermann endplate changes: disc space narrowing and facet arthropathy with vacuum phenomenon at L5/S1 on the CT of 5 August 2003, and multilevel lumbar disc degeneration and facet arthropathy on later imaging, so the definition is met.

The dictionary counts clinical onset back from that first confirming imaging to when symptoms became persistent; his back trouble after the 1980 injuries and chronic low back pain on Voltaren by October 1982 are consistent with the estimated onset of 7 January 1982.

Causative Factors — Balance of Probabilities (Statement of Principles concerning Thoracolumbar Spondylosis, No.

14 of 2023) Factor 9(1): having an inflammatory joint disease from the specified list of inflammatory joint diseases, in the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of ankylosing spondylitis, rheumatoid, psoriatic or reactive arthritis, or arthritis associated with inflammatory bowel disease; his sacroiliac joints were normal on the lumbosacral X-ray of 6 August 1980, and later imaging showed no sacroiliitis.

Factor 9(2): having non-viral infection of the affected joint, resulting in inflammation within that joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of infection of a lumbar facet joint or intervertebral disc before the estimated onset of 7 January 1982, and nothing in his history suggests it.

Factor 9(3): having an intra-articular fracture of the spine, at the level of, or adjacent to, the affected joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - The lumbosacral X-ray of 6 August 1980 showed no bone trauma, and the 1983 query of L5 spondylolysis, a pars interarticularis defect rather than an intra-articular fracture, was not confirmed; later MRI showed the partes intact.

Factor 9(4): having a spinal condition from the specified list of spinal conditions, affecting the thoracolumbar spine for at least the 6 months before the clinical onset of thoracolumbar spondylosis — MET - The specified list includes scoliosis and deformity of a vertebra.

The lumbosacral X-ray of 6 August 1980, 17 months before the estimated onset of 7 January 1982, showed a very minor scoliosis at L4 and L5 convex to the left, and a left-convex lumbar curve was again reported on the X-ray of 25 December 2010 and the MRI of 4 May 2011.

Scheuermann's disorder is not itself on the list, but its recorded sequelae, multiple endplate Schmorl's nodes in the lower thoracic and upper lumbar vertebrae, are deformities of vertebrae, and because the disorder develops during adolescent growth they were present well before onset.

These are constitutional factors, not related to service.

Factor 9(5): having undergone a spinal fusion, immediately above or below the affected joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran has never undergone spinal fusion; his only fusion operations were of the thumb and toe joints between 1980 and 1982.

Factor 9(6): for lumbar spondylosis only, having leg length inequality for at least the 5 years before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of a leg length inequality of at least 2 centimetres on examination or imaging, and nothing in his history suggests one.

Factor 9(7): having a depositional joint disease from the specified list of depositional joint diseases, in the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of gout, pseudogout, haemochromatosis, alkaptonuria or Wilson's disease, and nothing in his history suggests any of them.

Factor 9(8): having trauma to the thoracolumbar spine at least 6 months before the clinical onset of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical onset of thoracolumbar spondylosis — MET - the veteran sustained repeated discrete trauma to the lumbar spine more than six months before the estimated onset of 7 January 1982 and within the 20 years before it: a rugby scrum collapsed on top of him in organised sport on 26 November 1978 (tender lumbar spine, reduced range of movement); he strained his lower back lifting steel cables and cable drums at sea on 5 October 1979 (painful flexion and extension, then leg numbness); he fell directly onto his back at work on 5 August 1980 (sacral tenderness, straight leg raising limited to 50 degrees, right L4 root irritation); and a four-wheel-drive accident at Cooktown in late January 1981 was followed by back trouble 'on and off', as recorded in May 1983.

Factor 9(9): having a penetrating injury to an intervertebral disc before the clinical onset of thoracolumbar spondylosis at the level of, or adjacent to, the intervertebral disc injury — NOT MET - There is no record of a bullet, fragment, knife or needle piercing a lumbar intervertebral disc before the estimated onset, and nothing in his history suggests it.

Factor 9(10): having a thoracolumbar intervertebral disc prolapse at least 6 months before the clinical onset of thoracolumbar spondylosis at the level of the intervertebral disc prolapse — NOT MET - No disc prolapse was diagnosed before the estimated onset; the transient leg numbness of October 1979 and mild right L4 root irritation of August 1980 did not establish one, and herniation was first imaged on 5 August 2003.

Factor 9(11): lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine: (a) to a cumulative total of at least 150,000 kilograms within any 10 year period before the clinical onset of thoracolumbar spondylosis; and (b) where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period — MET - Load weights are not recorded, but heavy lifting was inherent in his duties as a Marine Technician and Marine Technician: working ships' lines and rope work, handling survey equipment, storing ship and, on 5 October 1979 in a frigate, lifting steel cables and cable drums, which strained his back.

Excluding his first year of recruit and category training, about 2.5 years of such duties preceded the estimated onset.

At a conservative 20 lifts of at least 25 kg on each of 150 heavy working days a year (allowing for leave, watchkeeping and post- operative light duties), the total estimated from his documented duties is about 187,500 kg (2.5 150 20 25), all within the 10 years immediately before onset.

Factor 9(12): carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine: (a) to a cumulative total of at least 3,800 hours within any 10 year period before the clinical onset of thoracolumbar spondylosis; and (b) where the clinical onset of thoracolumbar spondylosis occurred within the 25 years following that period — NOT MET - Carrying loads of at least 20 kg was intermittent in his duties; even at 30 minutes a day on 150 working days a year, his 3.5 years of service before onset give about 265 hours, far short of 3,800 hours.

Factor 9(13): for lumbar spondylosis only, flying in an engine powered aircraft as operational aircrew, for a cumulative total of at least 2,000 hours within the 25 years before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran served as a Marine Technician and Marine Technician and never flew as operational aircrew in any aircraft.

Factor 9(14): being obese for at least 10 years within the 25 years before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran was first recorded as obese (BMI 32.9) on 13 August 1981; even if he had been obese from soon after enlistment in July 1978, he could not have been obese for 10 years before the estimated onset of 7 January 1982.

Factor 9(15): having acromegaly involving the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of acromegaly or growth hormone excess at any time, and nothing in his history suggests it.

Factor 9(16): having Paget's disease of bone involving the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of Paget's disease of bone, and the later bone scans of 2011, 2015 and 2018 showed only degenerative facet and endplate uptake in the lumbar spine.

Causative Factors — Reasonable Hypothesis (Statement of Principles concerning Thoracolumbar Spondylosis, No.

13 of 2023) the veteran had no operational deployments.

The Reasonable Hypothesis standard applies only to operational service, so these factors are assessed for completeness and, like the Balance of Probabilities factors, by analogy only.

Factor 9(1): being a prisoner of war before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran was never a prisoner of war; he rendered peacetime naval service with no operational deployments.

Factor 9(2): having an inflammatory joint disease from the specified list of inflammatory joint diseases, in the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - No inflammatory joint disease on the specified list (ankylosing spondylitis, rheumatoid, psoriatic, reactive or enteropathic arthritis) is recorded, and his sacroiliac joints were normal on the lumbosacral X-ray of 6 August 1980.

Factor 9(3): having non-viral infection of the affected joint, resulting in inflammation within that joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of infection of a lumbar facet joint or disc before the estimated onset of 7 January 1982, and nothing in his history suggests it.

Factor 9(4): having an intra-articular fracture of the spine, at the level of, or adjacent to, the affected joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - No intra-articular spinal fracture is recorded: the X-ray of 6 August 1980 showed no bone trauma, and the 1983 query of L5 spondylolysis, a pars defect rather than an intra-articular fracture, was not confirmed on later MRI.

Factor 9(5): having a spinal condition from the specified list of spinal conditions, affecting the thoracolumbar spine for at least the 6 months before the clinical onset of thoracolumbar spondylosis — MET - The specified list includes scoliosis and deformity of a vertebra.

A very minor left-convex scoliosis at L4 and L5 was shown on the lumbosacral X-ray of 6 August 1980, 17 months before the estimated onset of 7 January 1982, and persisted on imaging in 2010 and 2011.

The multiple endplate Schmorl's nodes of Scheuermann's disorder, which develops during adolescent growth, are deformities of vertebrae that were present well before onset.

These are constitutional factors, not related to service.

Factor 9(6): for lumbar spondylosis only, having a kyphotic abnormality or lordotic abnormality affecting the lumbar spine for at least the 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - No abnormal lumbar kyphosis or lordosis is recorded; the X-ray of 25 December 2010 reported satisfactory sagittal alignment and the MRI of 10 April 2014 normal alignment, his scoliosis being a lateral rather than sagittal curve.

Factor 9(7): having undergone a spinal fusion, immediately above or below the affected joint, at least 6 months before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran has never undergone spinal fusion; his fusion operations were confined to the thumb and toe joints.

Factor 9(8): for lumbar spondylosis only, having leg length inequality for at least the 2 years before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of a leg length inequality of at least 2 centimetres, and nothing in his history suggests one.

Factor 9(9): having a depositional joint disease from the specified list of depositional joint diseases, in the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of gout, pseudogout, haemochromatosis, alkaptonuria or Wilson's disease, and nothing in his history suggests any of them.

Factor 9(10): having trauma to the thoracolumbar spine at least 6 months before the clinical onset of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical onset of thoracolumbar spondylosis — MET - More than six months before the estimated onset of 7 January 1982, and within 20 years of it, the veteran sustained repeated discrete lumbar trauma: a rugby scrum collapse in organised sport on 26 November 1978 (tender lumbar spine, reduced movement); a strain lifting steel cables and cable drums at sea on 5 October 1979 (painful movement, leg numbness); a direct fall onto his back at work on 5 August 1980 (sacral tenderness, straight leg raising limited to 50 degrees, right L4 root irritation); and a four-wheel-drive accident at Cooktown in late January 1981, after which he had back trouble on and off.

Factor 9(11): having a penetrating injury to an intervertebral disc before the clinical onset of thoracolumbar spondylosis at the level of, or adjacent to, the intervertebral disc injury — NOT MET - There is no record of any object piercing a lumbar intervertebral disc before the estimated onset, and nothing in his history suggests it.

Factor 9(12): having a thoracolumbar intervertebral disc prolapse at least 6 months before the clinical onset of thoracolumbar spondylosis at the level of the intervertebral disc prolapse — NOT MET - No disc prolapse was diagnosed before the estimated onset; the transient leg numbness of October 1979 and right L4 root irritation of August 1980 did not establish one, and herniation was first imaged in August 2003.

Factor 9(13): lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 100,000 kilograms within any 10 year period before the clinical onset of thoracolumbar spondylosis — MET - Heavy lifting was inherent in his documented duties as a Marine Technician and Marine Technician, including rope work, survey equipment and, on 5 October 1979, steel cables and cable drums that strained his back.

Estimated from those duties, about 2.5 years of such work before the estimated onset (excluding recruit and category training), at a conservative 20 lifts of at least 25 kg on 150 heavy working days a year, totals about 187,500 kg (2.5 150 20 25), well above 100,000 kg within the 10 years before onset.

Factor 9(14): carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 3,800 hours within any 10 year period before the clinical onset of thoracolumbar spondylosis — NOT MET - Carrying loads of at least 20 kg was intermittent; even 30 minutes a day on 150 working days a year over his 3.5 years of service before onset gives about 265 hours, far short of 3,800 hours.

Factor 9(15): for lumbar spondylosis only, flying in an engine powered aircraft as operational aircrew, for a cumulative total of at least 1,000 hours within the 25 years before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran served as a Marine Technician and Marine Technician and never flew as operational aircrew in any aircraft.

Factor 9(16): being obese for at least 10 years before the clinical onset of thoracolumbar spondylosis — NOT MET - the veteran was first recorded as obese (BMI 32.9) on 13 August 1981, less than five months before the estimated onset of 7 January 1982, so he had not been obese for 10 years before it.

Factor 9(17): having acromegaly involving the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of acromegaly or growth hormone excess at any time, and nothing in his history suggests it.

Factor 9(18): having Paget's disease of bone involving the thoracolumbar spine before the clinical onset of thoracolumbar spondylosis — NOT MET - There is no record of Paget's disease of bone, and later bone scans showed only degenerative uptake in the lumbar spine.

Other Plausible Links to Service Independently of the Statement of Principles, the veteran's lumbar symptoms began with injuries that arose out of, or in the course of, his employment as a member.

At a frigate, as a young adult, he strained his lower back after a heavy rugby tackle in October 1978, and on 26 November 1978 a scrum collapsed on top of him during organised sport on duty; on 5 October 1979 he strained his back lifting steel cables and cable drums during survey work at sea in a frigate; on 5 August 1980 he fell directly onto his back at work, with right L4 nerve root irritation; and on 29 April 1983 he slipped on soapy water while scrubbing a deck on duty and landed on the floor polisher, injuring his back.

The spondylosis can also be regarded as a sequela of these in-service lumbar injuries.

The records show continuity: back trouble 'on and off' after the 1980 injuries, chronic low back pain treated with Voltaren by October 1982, and 'recurrent' low back pain with tenderness from L4 to S1 requiring physiotherapy and orthopaedic review in February and June 1983.

The first post-service imaging, the CT of 5 August 2003, showed established degeneration at the same segments: disc space narrowing, disc herniation and facet arthropathy with vacuum phenomenon at L5/S1, and a disc protrusion at L4/5.

Under the DRCA, a disease is compensable where employment contributed to it to a significant degree, and repeated injury to these segments in service is such a contribution.

Service also aggravated a constitutional predisposition.

His minor left-convex lumbar scoliosis (first recorded on 6 August 1980) and Scheuermann's endplate changes are not due to service, but they leave the spine more vulnerable to trauma and loading.

After his back pain became chronic he continued Marine Technician and ships' husbandry duties; in May 1983 he was restricted to light duties with no lifting or driving, yet heavy lifting is again recorded at a frigate in October 1984, and his back was never investigated beyond plain X-rays before discharge.

Continued heavy loading of a symptomatic, uninvestigated spine is an aggravation of the condition by his employment.

Conclusion the veteran's lumbar spondylosis arose from repeated trauma to the lumbar spine and cumulative heavy lifting during his naval service, acting on a spine with a minor constitutional scoliosis and Scheuermann's endplate changes.

The in-service injuries of 1978 to 1980 and an estimated 187,500 kg of heavy lifting before onset meet the trauma and lifting factors, and the progression to chronic low back pain treated with anti-inflammatory medication by 1982 shows the condition developing in service; the scoliosis and vertebral deformity are a background factor unrelated to service.

After onset, further in-service trauma, continued heavy duties and the absence of investigation worsened the condition, which was later aggravated by long-standing obesity and the post-service fall of April 2011.

Service contributed to its onset and progression to a significant degree.

The % contribution of the causes is 100% and significant.

Worsening Factors In the alternative, if the veteran's lumbar spondylosis is found not to have arisen out of his service, the following factors address whether service rendered after its clinical onset (estimated 7 January 1982) aggravated it or contributed to it in a material degree.

He served for a further three years and three months after that date, during which he was injured when a car slid off its jack (September 1982) and in a fall onto a floor polisher on duty (May 1983), and continued heavy duties while taking anti-inflammatory medication for chronic low back pain; post-service factors are also addressed.

Worsening Factors — Balance of Probabilities (Statement of Principles concerning Thoracolumbar Spondylosis, No.

14 of 2023) Factor 9(17): having an inflammatory joint disease from the specified list of inflammatory joint diseases, in the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - No inflammatory joint disease on the specified list has been diagnosed at any time, and imaging from 2014 to 2018 showed no sacroiliitis or erosive arthropathy.

Factor 9(18): having non-viral infection of the affected joint, resulting in inflammation within that joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - No infection of a lumbar facet joint or disc has been recorded at any time, including after the facet joint injection of 19 August 2011 and the rhizotomies of 2019.

Factor 9(19): having an intra-articular fracture of the spine, at the level of, or adjacent to, the affected joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - No intra-articular fracture at or adjacent to the affected lumbar levels preceded the recorded worsening: the 1983 query of L5 spondylolysis was not confirmed, no fracture was found after the 2011 fall, and a chronic T12 endplate depression was first reported only in February 2020.

Factor 9(20): having a spinal condition from the specified list of spinal conditions, affecting the thoracolumbar spine for at least the 6 months before the clinical worsening of thoracolumbar spondylosis — MET - the veteran's constitutional spinal deformities from the specified list persisted after onset: a left-convex lumbar scoliosis was recorded in 1980 and again in 2010 and 2011, the multiple endplate Schmorl's nodes of Scheuermann's disorder are permanent deformities of the vertebrae, and developmentally asymmetrical posterior elements at L5/S1 were described in 2016.

These affected the thoracolumbar spine for decades before the measurable worsening from the CT of 5 August 2003 (L5/S1 disc herniation and facet arthropathy) to the MRI of 29 January 2020 (high-grade L5/S1 facet degeneration, synovial cyst and retrolisthesis).

They are not related to service.

Factor 9(21): having undergone a spinal fusion, immediately above or below the affected joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - the veteran has never undergone spinal fusion; his fusion operations were confined to the thumb and toe joints between 1980 and 1982.

Factor 9(22): for lumbar spondylosis only, having leg length inequality for at least the 5 years before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of a leg length inequality of at least 2 centimetres at any time, and nothing in his history suggests one.

Factor 9(23): having trauma to the thoracolumbar spine at least 6 months before the clinical worsening of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical worsening of thoracolumbar spondylosis — MET - After onset, on 17 September 1982, a car slid off its jack and pinned him, causing low back pain radiating down the left leg with straight leg raising limited to 70 degrees; more than six months later, in February and June 1983, his 'recurrent' low back pain with limited movement required lumbar X-rays, physiotherapy, orthopaedic referral and light duties with no lifting, following a further fall onto a floor polisher on duty on 29 April 1983.

After service, a workplace fall onto his back on 2 April 2011, with months of pain and a right L4/5 facet injection, preceded by over six months the new left L5/S1 synovial cyst of April 2014 and later denervation and retrolisthesis, all within 20 years.

Factor 9(24): having a depositional joint disease from the specified list of depositional joint diseases, in the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of gout, pseudogout, haemochromatosis, alkaptonuria or Wilson's disease at any time, and nothing in his history suggests any of them.

Factor 9(25): having a penetrating injury to an intervertebral disc before the clinical worsening of thoracolumbar spondylosis at the level of, or adjacent to, the intervertebral disc injury — NOT MET - No lumbar disc has been pierced by a needle or other object: the CT-guided injection of 19 August 2011 was into the right L4/5 facet joint, the 2019 rhizotomies targeted the facet joints, and no discography is recorded.

Factor 9(26): having a thoracolumbar intervertebral disc prolapse at least 6 months before the clinical worsening of thoracolumbar spondylosis at the level of the intervertebral disc prolapse — MET - The CT of 5 August 2003, performed for acute right L5/S1 nerve root pain, showed a right posterolateral L5/S1 disc herniation impinging on the right L5 nerve root (a sequestrated fragment could not be excluded) and a L4/5 disc protrusion.

More than six months later those levels worsened measurably: moderate L5/S1 disc degeneration with a disc-osteophyte complex and left facet arthropathy (2011), a new left L5/S1 facet synovial cyst (2014), left sacral paraspinal denervation (2015), left L5 foraminal impingement (2016 and 2017), and a 4 mm L5/S1 retrolisthesis with a shallow L4/5 disc extrusion (2019).

This factor is met on post-service facts.

Factor 9(27): lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine: (a) to a cumulative total of at least 150,000 kilograms within any 10 year period before the clinical worsening of thoracolumbar spondylosis; and (b) where the clinical worsening of thoracolumbar spondylosis occurred within the 25 years following that period — MET - After onset the veteran served a further three years and three months: at sea in a frigate, in ships' husbandry at the Fleet Intermediate Maintenance Activity, Sydney (including scrubbing and polishing decks), and at a frigate, where 'heavy lifting' is recorded in October 1984.

Allowing for about four months of ineffective or restricted duty, about 2.9 years at a conservative 20 lifts of at least 25 kg on 150 heavy working days a year gives an estimated 217,500 kg (2.9 150 20 25) from his documented duties, and about 400,000 kg over his whole service, all within one 10-year period.

The worsening shown on the CT of 5 August 2003 and on imaging in 2010 and 2011 followed within 25 years of that period.

Factor 9(28): carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine: (a) to a cumulative total of at least 3,800 hours within any 10 year period before the clinical worsening of thoracolumbar spondylosis; and (b) where the clinical worsening of thoracolumbar spondylosis occurred within the 25 years following that period — NOT MET - Carrying loads of at least 20 kg was intermittent; even 30 minutes a day on 150 working days a year over his whole 6.8 years of service gives about 510 hours, and no heavy post- service carrying is recorded.

Factor 9(29): for lumbar spondylosis only, flying in an engine powered aircraft as operational aircrew, for a cumulative total of at least 2,000 hours within the 25 years before the clinical worsening of thoracolumbar spondylosis — NOT MET - the veteran served as a Marine Technician and Marine Technician and never flew as operational aircrew in any aircraft.

Factor 9(30): being obese for at least 10 years within the 25 years before the clinical worsening of thoracolumbar spondylosis — MET - the veteran was obese through most of 1981 to 1985 (BMI 32.9 in August 1981 and 35.7 at discharge in April 1985), and the later records indicate that he remained so: his body habitus made ultrasound technically very difficult in 2011, he weighed about 168 kg (BMI about 48.6) before bariatric surgery in 2013, and his BMI was 41.5 on 19 December 2018.

He was therefore obese for well over 10 years within the 25 years before the worsening documented from 2003 to 2019, including the new synovial cyst (2014), denervation (2015) and retrolisthesis (2019).

This factor is met largely on post-service facts, although the obesity began during service.

Factor 9(31): having acromegaly involving the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of acromegaly or growth hormone excess at any time, and nothing in his history suggests it.

Factor 9(32): having Paget's disease of bone involving the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of Paget's disease of bone; the bone scans of 2011, 2015 and 2018 showed only degenerative facet and endplate uptake.

Factor 9(33): inability to obtain appropriate clinical management for thoracolumbar spondylosis — MET - the veteran presented repeatedly with lumbar symptoms from 1978, with nerve root signs and chronic low back pain on Voltaren by 1982, yet had only anti-inflammatories, brief light duties and one plain X-ray before May 1983; the queried L5 spondylolysis was then not pursued, and his discharge examination did not mention his back.

Under Brew v Repatriation Commission (Full Federal Court, 10 September 1999, Merkel J), inability is assessed objectively and subjectively and includes psychological or emotional incapacity and the threat of sanctions; his barriers included treatment at sea without a medical officer (1979), remote care at Cooktown (1980), the August 1981 signal suspecting him of malingering and the loss of his medical record (1984).

Diagnosis came only with the CT of 5 August 2003, almost 25 years after first presentation, when disc herniation was established.

Worsening Factors — Reasonable Hypothesis (Statement of Principles concerning Thoracolumbar Spondylosis, No.

13 of 2023) the veteran had no operational deployments.

The Reasonable Hypothesis standard applies only to operational service, so these factors are assessed for completeness and, like the Balance of Probabilities factors, by analogy only.

Factor 9(19): being a prisoner of war before the clinical worsening of thoracolumbar spondylosis — NOT MET - the veteran was never a prisoner of war at any time; he rendered peacetime naval service with no operational deployments.

Factor 9(20): having an inflammatory joint disease from the specified list of inflammatory joint diseases, in the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - No inflammatory joint disease on the specified list has been diagnosed at any time, and imaging from 2014 to 2018 showed no sacroiliitis.

Factor 9(21): having non-viral infection of the affected joint, resulting in inflammation within that joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - No infection of a lumbar facet joint or disc has been recorded, including after the facet joint injection of 19 August 2011 and the 2019 rhizotomies.

Factor 9(22): having an intra-articular fracture of the spine, at the level of, or adjacent to, the affected joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - No intra-articular fracture at or adjacent to the affected lumbar levels preceded the recorded worsening; the 1983 query of L5 spondylolysis was not confirmed, no fracture was found after the 2011 fall, and a T12 endplate depression was first reported only in February 2020.

Factor 9(23): having a spinal condition from the specified list of spinal conditions, affecting the thoracolumbar spine for at least the 6 months before the clinical worsening of thoracolumbar spondylosis — MET - The specified list includes scoliosis and deformity of a vertebra.

The veteran's left-convex lumbar scoliosis (recorded in 1980, 2010 and 2011), the permanent endplate deformities of Scheuermann's disorder with multiple Schmorl's nodes, and the developmentally asymmetrical L5/S1 posterior elements described in 2016 affected his thoracolumbar spine for decades before the worsening documented from the CT of 5 August 2003 to the MRI of 29 January 2020.

These constitutional factors are not related to service.

Factor 9(24): for lumbar spondylosis only, having a kyphotic abnormality or lordotic abnormality affecting the lumbar spine for at least the 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - No abnormal lumbar kyphosis or lordosis is recorded; the X-ray of 25 December 2010 reported satisfactory sagittal alignment and the MRI of 10 April 2014 normal vertebral alignment.

Factor 9(25): having undergone a spinal fusion, immediately above or below the affected joint, at least 6 months before the clinical worsening of thoracolumbar spondylosis — NOT MET - the veteran has never undergone spinal fusion; his fusion operations were confined to the thumb and toe joints.

Factor 9(26): for lumbar spondylosis only, having leg length inequality for at least the 2 years before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of a leg length inequality of at least 2 centimetres at any time, and nothing in his history suggests one.

Factor 9(27): having a depositional joint disease from the specified list of depositional joint diseases, in the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of gout, pseudogout, haemochromatosis, alkaptonuria or Wilson's disease, and nothing in his history suggests any of them.

Factor 9(28): having trauma to the thoracolumbar spine at least 6 months before the clinical worsening of thoracolumbar spondylosis, and where the trauma to the thoracolumbar spine occurred within the 20 years before the clinical worsening of thoracolumbar spondylosis — MET - On 17 September 1982, after onset, a car slid off its jack and pinned him, causing low back pain radiating down the left leg with limited straight leg raising; more than six months later, in February and June 1983, his recurrent low back pain with limited movement required X- rays, physiotherapy, orthopaedic referral and light duties with no lifting.

After service, a workplace fall onto his back on 2 April 2011, with months of pain and a right L4/5 facet injection, preceded the new left L5/S1 synovial cyst of April 2014 and the later denervation and retrolisthesis by more than six months, all within 20 years.

Factor 9(29): having a penetrating injury to an intervertebral disc before the clinical worsening of thoracolumbar spondylosis at the level of, or adjacent to, the intervertebral disc injury — NOT MET - No lumbar disc has been pierced by a needle or other object; the 2011 injection was into the right L4/5 facet joint, the 2019 rhizotomies targeted the facet joints, and no discography is recorded.

Factor 9(30): having a thoracolumbar intervertebral disc prolapse at least 6 months before the clinical worsening of thoracolumbar spondylosis at the level of the intervertebral disc prolapse — MET - The CT of 5 August 2003 showed a right L5/S1 disc herniation impinging on the right L5 nerve root and a L4/5 disc protrusion.

More than six months later those levels worsened measurably, with a new left L5/S1 facet synovial cyst (2014), left sacral paraspinal denervation (2015), left L5 foraminal impingement (2016 and 2017), and a 4 mm L5/S1 retrolisthesis with a shallow L4/5 disc extrusion (2019).

This factor is met on post-service facts.

Factor 9(31): lifting loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 100,000 kilograms within any 10 year period before the clinical worsening of thoracolumbar spondylosis — MET - After onset the veteran served a further three years and three months in a frigate, in ships' husbandry at the Fleet Intermediate Maintenance Activity, Sydney, and at a frigate, where heavy lifting is recorded in October 1984.

Estimated from those duties, about 2.9 effective years at a conservative 20 lifts of at least 25 kg on 150 heavy working days a year gives about 217,500 kg (2.9 150 20 25), and about 400,000 kg over his whole service, well above 100,000 kg within a 10-year period before the worsening documented from 2003.

Factor 9(32): carrying loads of at least 20 kilograms while bearing the weight through the thoracolumbar spine to a cumulative total of at least 3,800 hours within any 10 year period before the clinical worsening of thoracolumbar spondylosis — NOT MET - Carrying loads of at least 20 kg was intermittent; even 30 minutes a day on 150 working days a year over his 6.8 years of service gives about 510 hours, far short of 3,800 hours.

Factor 9(33): for lumbar spondylosis only, flying in an engine powered aircraft as operational aircrew, for a cumulative total of at least 1,000 hours within the 25 years before the clinical worsening of thoracolumbar spondylosis — NOT MET - the veteran served as a Marine Technician and Marine Technician and never flew as operational aircrew in any aircraft.

Factor 9(34): being obese for at least 10 years before the clinical worsening of thoracolumbar spondylosis — MET - the veteran was obese through most of 1981 to 1985 (BMI 32.9 in August 1981 and 35.7 at discharge in April 1985), and later records indicate that he remained so, weighing about 168 kg (BMI about 48.6) before bariatric surgery in 2013 and with a BMI of 41.5 in December 2018.

He was therefore obese for well over 10 years before the worsening documented from 2003 to 2019.

This factor is met largely on post-service facts, although the obesity began during service.

Factor 9(35): having acromegaly involving the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of acromegaly or growth hormone excess at any time, and nothing in his history suggests it.

Factor 9(36): having Paget's disease of bone involving the thoracolumbar spine before the clinical worsening of thoracolumbar spondylosis — NOT MET - There is no record of Paget's disease of bone; the bone scans of 2011, 2015 and 2018 showed only degenerative uptake.

Factor 9(37): inability to obtain appropriate clinical management for thoracolumbar spondylosis — MET - the veteran's recurrent in-service lumbar presentations from 1978, including nerve root signs and chronic low back pain on Voltaren by 1982, were managed with anti-inflammatories and light duties, without specialist referral until May 1983 or investigation beyond plain X- rays, and the condition was not diagnosed until the CT of 5 August 2003, almost 25 years later.

Under Brew v Repatriation Commission (Full Federal Court, 10 September 1999, Merkel J), inability is assessed objectively and subjectively and includes psychological or emotional incapacity and the threat of sanctions; his barriers included sea and remote postings, the August 1981 signal suspecting him of malingering and the loss of his medical record in 1984

Sequelae

Lumbar spondylosis is in part a sequela of the veteran's in-service lumbar injuries: the lumbar strains of October 1978 and October 1979, the lumbar sprain of December 1978 and the lumbar soft tissue injuries of 1980, 1982 and 1983.

These injuries, together with cumulative heavy lifting in service, initiated and accelerated a degenerative process in a spine that also had a minor constitutional scoliosis and Scheuermann's endplate changes, and it has progressed to the current advanced multilevel lumbar degeneration

Unintended Consequence

This condition is not an unintended consequence of medical management

Inability to Attain Appropriate Medical Management

This only applies to worsening factors.

The veteran presented repeatedly with lumbar symptoms from October 1978, including leg numbness (October 1979), right L4 nerve root irritation (August 1980) and chronic low back pain treated with Voltaren (October 1982), but was managed with anti-inflammatory medication, brief periods of light duty and a single plain X-ray (August 1980) until physiotherapy, further X-rays and orthopaedic referral in May 1983.

The queried L5 spondylolysis was not investigated further, his discharge examination made no mention of his back, and the condition was not diagnosed until the CT of 5 August 2003, almost 25 years after his first presentation, when a disc herniation impinging on the right L5 nerve root was already established — MET.

Under Brew v Repatriation Commission (Full Federal Court, 10 September 1999, Merkel J), inability to obtain appropriate clinical management is assessed objectively and subjectively and includes psychological or emotional incapacity and the threat of sanctions that make seeking treatment unrealistic.

The veteran's documented barriers included treatment at sea by a Marine Technician medical assistant in October 1979, with review by a medical officer deferred until the ship returned to a frigate, remote care at Cooktown after the 1980 accident, the command signal of 18 August 1981 suspecting him of malingering, and the loss of his PM168 medical record at a frigate in May 1984

Date of Clinical Onset

Lumbar spondylosis is a chronic condition.

Its date of clinical onset is the date on which the condition first manifested clinically.

It is not the date on which the condition was first imaged or formally diagnosed

Imaging

And specialist diagnosis record when an established condition was confirmed; a chronic condition is present, and usually symptomatic, for a considerable period before it is investigated, so the date of imaging must not be taken as the date of clinical onset.

First documented presentation with pain in the lumbar spine: 4 October 1978 — lumbar back strain at a frigate, with aching of the back on parade three days after a heavy rugby tackle and lumbar tenderness on examination [CHART REVIEW document].

End of ADF service: 13 April 1985.

First imaging / formal diagnosis (confirmation, not onset): 5 August 2003 (CT of the lumbosacral spine).

The precise date of clinical onset cannot be determined from the records.

It is therefore estimated as the midpoint between the first documented presentation with pain in the lumbar spine (4 October 1978) and the end of the veteran's ADF service (13 April 1985).

Estimated date of clinical onset: 7 January 1982.

This date falls within the veteran's ADF service (28 June 1978 13 April 1985).

In-service events and exposures before this date relate to clinical onset, and those after it to clinical worsening

Part of DVA Claims — Back & neck — every Statement of Principles covering the back and neck.

Others in this area:

Dr Thomas Perkins, Veterans Health Centre

Reviewed by Dr Thomas Perkins

Founding doctor, Veterans Health Centre · former RAAF aviation medical officer · 13 years working exclusively with ADF members and veterans. Full profile →